{"id":144875,"date":"2026-06-06T12:32:27","date_gmt":"2026-06-06T15:32:27","guid":{"rendered":"https:\/\/ubirataonline.com.br\/yale-study-links-some-long-covid-patients-to-autoimmune-responses\/"},"modified":"2026-06-06T12:32:28","modified_gmt":"2026-06-06T15:32:28","slug":"yale-study-links-some-long-covid-patients-to-autoimmune-responses","status":"publish","type":"post","link":"https:\/\/ubirataonline.com.br\/en\/yale-study-links-some-long-covid-patients-to-autoimmune-responses\/","title":{"rendered":"Yale study links some long COVID patients to autoimmune responses"},"content":{"rendered":"

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\u201cThis is a significant finding, but that\u00a0doesn\u2019t mean there aren\u2019t other causes,\u201d said Iwasaki, who has been\u00a0a leading investigator on COVID<\/span> immune response. \u201cOur study does not explain the entire long COVID<\/span> scenario. This is one possible cause of long COVID<\/span>, but it will likely have other trigger causes as\u00a0well.\u201d<\/p>\n

Still, if eventually validated, the study could point toward future long COVID<\/span> treatments, including therapies already used for other autoimmune diseases, although the researchers stress that much more investigation is\u00a0needed.<\/p>\n

Iwasaki is also a professor of dermatology and of molecular, cellular, and developmental biology in Yale\u2019s Faculty of Arts and Sciences, a professor of epidemiology at Yale School of Public Health, and an investigator of the Howard Hughes Medical\u00a0Institute.<\/p>\n

Since long COVID<\/span> first emerged six years ago, researchers in Iwasaki\u2019s lab have been chipping away at the mysterious condition. Its causes have been difficult to pin down because symptoms vary widely from patient to patient. But the new findings provide some of the strongest evidence so far that autoimmunity may be one important\u00a0factor.<\/p>\n

For the study, the Yale team and collaborators \u2014 including a Mount Sinai Health System team led by study co-senior author David Putrino, a professor of rehabilitation and human performance and the Nash Family Director of the Cohen Center for Recovery from Complex Chronic Illness \u2014 focused specifically on autoantibodies, immune proteins that are known to mistakenly target the body\u2019s own tissues instead of viruses or\u00a0bacteria.<\/p>\n

The researchers discovered that many long COVID<\/span> patients had autoantibodies aimed at parts of the brain and nervous\u00a0system.\u00a0<\/p>\n

These autoantibodies frequently targeted tissues involved in pain signaling, memory, balance, sensory processing, and autonomic nervous system control, they found, which could help explain long COVID<\/span> symptoms such as brain fog, dizziness, headaches, fatigue, burning pain, and\u00a0numbness.<\/p>\n

After analyzing blood samples from people with long COVID<\/span>, from healthy volunteers, and from people who had recovered from COVID<\/span> but had not developed lasting symptoms, the researchers purified antibodies from patients\u2019 blood and exposed them to human and mouse tissues. They found that antibodies from long COVID<\/span> patients reacted more strongly with certain brain regions and nerve tissues than antibodies from control\u00a0groups.<\/p>\n

The researchers then screened these blood samples against more than 21,000 human proteins to identify what the antibodies were targeting. Many of the targets, they found, were linked to neurons, nerve communication, inflammation, and hormone\u00a0signaling.<\/p>\n

Finally, they transferred antibodies from long COVID<\/span> patients into healthy mice. The researchers conducted various behavioral studies\u00a0working with a team led by study co-senior author Tamas Horvath, the Jean and David W. Wallace Professor of Comparative Medicine, professor of neuroscience and of obstetrics, gynecology, and reproductive sciences at YSM<\/span>.\u00a0They observed that the mice developed increased pain sensitivity, fatigue, impaired balance, and damage to small nerve\u00a0fibers.\u00a0<\/p>\n

Working with Marc Schneeberger Pane, assistant professor in cellular and molecular physiology in Yale\u2019s Faculty of Arts and Sciences,the team found that mice also showed abnormal neuronal activation in brain regions involved in pain, fatigue, memory, and emotional\u00a0regulation.<\/p>\n

\u201cWhat was most fascinating about this work is that we were able to find antibodies that, when transferred to mice, caused the same type of symptoms that are reported by long COVID<\/span> patients,\u201d said study lead author\u00a0Keyla Santos Guedes de S\u00e1, a postdoctoral associate in immunobiology in Iwasaki\u2019s\u00a0lab.\u00a0<\/p>\n

Researchers will next examine how the damage is being\u00a0done.<\/p>\n

\u201cNow that we were able to identify a subgroup of patients whose condition might be driven by autoantibodies, we want to investigate the neurological and immunological mechanisms by which these autoantibodies are causing disease,\u201d said\u00a0S\u00e1.\u00a0\u00a0<\/p>\n

The rise of long COVID<\/span> itself wasn\u2019t a surprise, Iwasaki\u00a0added.<\/p>\n

\u201cWhen you dig into the literature, you see that every major pandemic is accompanied by a long-version, chronic illness that follows,\u201d she said. \u201cIt happens when a human population is exposed to a new pathogen \u2014 or sometimes not even a new pathogen but an existing one like EBV<\/span> [Epstein-Barr virus.] Many viral pathogens are capable of triggering these chronic diseases after\u00a0infection.\u201d<\/p>\n

For researchers, discovering effective treatments is the end\u00a0goal.\u00a0<\/p>\n

\u201cWe\u2019re all \u2014 Keyla and I and everybody in our team \u2014 driven by the desire to help people with long COVID<\/span>,\u201d Iwasaki said. \u201cRight now, there is no approved treatment for these people, and they really need\u00a0help.\u201d\u00a0<\/p>\n

Other Yale researchers involved in the study included scientists from the Department of Immunobiology in YSM<\/span>, the Center for Infection and Immunity, the Department of Comparative Medicine, the Department of Cellular and Molecular Physiology, and the Department of Pathology. Other partners included researchers from the Yale Cancer Biology Institute on West Campus and from the Department of Biostatistics at the Yale School of Public\u00a0Health.\u00a0<\/p>\n

In addition to scientists from the Icahn School of Medicine at Mount Sinai, other partners included researchers from the Howard Hughes Medical Institute; Serom Yx Systems; CellTrend GmbH in Germany; and the Institute of Medical Immunology, Charit\u00e9 – Universit\u00e4tsmedizin Berlin, Corporate Member of Freie Universit\u00e4t Berlin and Humboldt Universit\u00e4t zu Berlin and Berlin Institute of\u00a0Health.<\/p>\n<\/div>\n

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\u201cThis is a significant finding, but that\u00a0doesn\u2019t mean there aren\u2019t other causes,\u201d said Iwasaki, who has been\u00a0a leading investigator on COVID immune response. \u201cOur study does not explain the entire long COVID scenario. This is one possible cause of long COVID, but it will likely have other trigger causes as\u00a0well.\u201d Still, if eventually validated, the […]<\/p>\n","protected":false},"author":1,"featured_media":144876,"comment_status":"closed","ping_status":"","sticky":false,"template":"","format":"standard","meta":{"googlesitekit_rrm_CAow2JLhCw:productID":"","_gspb_post_css":"","maa_idioma":"","maa_pais":"","footnotes":""},"categories":[1],"tags":[40216,15652,13861,10762,10662,40217,10716,10747],"class_list":["post-144875","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-ubirata","tag-autoimmune","tag-covid","tag-links","tag-long","tag-patients","tag-responses","tag-study","tag-yale"],"blocksy_meta":[],"yoast_head":"\nYale study links some long COVID patients to autoimmune responses - Ubirat\u00e3 Online Not\u00edcias - A realidade ao seu alcance!<\/title>\n<meta name=\"description\" content=\"A new study co-led by Yale scientists reveals that antibodies from some long COVID patients attacked brain and nerve tissues, a finding that could point toward treatments.\" \/>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/ubirataonline.com.br\/en\/yale-study-links-some-long-covid-patients-to-autoimmune-responses\/\" \/>\n<meta property=\"og:locale\" content=\"en_US\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"Yale study links some long COVID patients to autoimmune responses\" \/>\n<meta property=\"og:description\" content=\"A new study co-led by Yale scientists reveals that antibodies from some long COVID patients attacked brain and nerve tissues, a finding that could point toward treatments.\" \/>\n<meta property=\"og:url\" content=\"https:\/\/ubirataonline.com.br\/en\/yale-study-links-some-long-covid-patients-to-autoimmune-responses\/\" \/>\n<meta property=\"og:site_name\" content=\"Ubirat\u00e3 Online Not\u00edcias - 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